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home > slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation > slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation
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Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link SLC25A39 binds and modulates PRDX1 to suppress ROS induced necroptosis in hepatocellular carcinoma ScienceDirect Frontiers Regulation of Heme Synthesis by Mitochondrial Homeostasis Proteins Mitochondrial Dysfunction, Transfer, and Therapeutic Strategies in Neurological Disorders ScienceOpen Up regulated SLC25A39 promotes cell growth and metastasis via regulating ROS production in colorectal cancer
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease  Manifestation
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease  Manifestation
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease  Manifestation
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease  Manifestation
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease  Manifestation

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

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